• Keine Ergebnisse gefunden

Literatur

Im Dokument 1.2.1 Morbus Crohn (Seite 66-74)

[1] Junqueira LC, Carneiro J, Kelley RO, Gratzl M.

Histologie.

Springer Verlag, Berlin, 2000

[2] Lehnert H, Werdan K.

Innere Medizin-essentials . Thieme Verlag, Stuttgart, 2006

[3] www.miami.uni-muenster.de/servelets/Derivate Servlet/ Derivate- 2052/02_arbeiten.pdf

gesehen am 26.11.2007

[4] Böcker W, Denk H, Heitz HP.

Pathologie.

Urban & Fischer Verlag, München, Jena, April 2004

[5] Schölmerich J.

Chronisch entzündliche Darmerkrankungen am Ende ihres ersten Jahrhunderts.

W. Zuckerschwerdt Verlag, München, 2001

[6] Picco MF, Bayless TM.

Tobacco Consumption and Disease Duration Are

Associat With Fistulizing and Stricturing Behaviors in the First 8 Years of Crohn`s Disease.

Am Journal Gastroenterol, 98:363-368, 2003.

[7] Regueiro M, Kip KE, Cheung O, Hegazi RA, Plevy S.

Cigarette smoking and age at diagnosis of inflammatory bowel disease.

Inflamm Bowel Dis, 11:42-47, 2005.

[8] Karlinger K, Györke T, Makö E, Mester A, Tarjan Z.

The epidemiology and the pathogenesis of inflammatory bowel disease.

Europ J Radiology, 35:154-167, 2000.

[9] Koutrobakis I, Manousos ON, Meuwissen SG, Pena AS.

Environmental risk factors in inflammatory bowel disease.

Hepatogastroenterology, 43:381-393, 1996

[10] Danese S, Fiocchi C.

Etiopathogenesis of inflammatory bowel diseaes.

World J Gastroenterol, 14:4807-4812.

[11] Bonen DK, Cho JH.

The Genetics of Inflammatory Bowel Disease.

Gastroenterology,124:521-536, 2003

[12] Hugot JP,Cho JH.

Updates on genetics of inflammatory bowel disease.

Curr Opinion Gastroenterol., 18:410-415, 2002

[13] Romagnani P, Annunziato F, Baccari MC, Parronchi P.

T cells and cytokines in Crohn`s disease.

Curr Opinion Immun. 9:793-799, 1997

[14] Maeda S, Hsu LC, Liu H, Bankston LA, Iimura M, Kagnoff M, Eckmann L, Karin M.

Nod2 Mutation in Crohn`s Disease Potentiates NF-ĸB Activity and IL-1β Processing.

Science,307:734-738, 2005

[15] Janeway CA, Travers D, Walport M, Shlomchik M.

Immunology.

Spektrum AkademischerVerlag GmbH, Heidelberg, Berlin, 2002

[16] Smith PD, Ochsenbauer-Jambor C, Smythies LE.

Intestinal macrophages: unique effector cells of the innate immune system.

Immunological Reviews, 206:149-159, 2005

[17] Spöttl T, Hausmann M, Kreutz M, Peuker A, Vogl D, Schölmerich J, Falk W, Andreesen R, Andus T, Herfarth H, Rogler G.

Monocyte differentiation in intestine-like macrophage phenotype induced by epthelial cells.

Journal of Leukozyte Biology, 70: 241-251, 2001

[18] Hausmann M, Bataille F, Spöttl T, Schreiter K, Falk W, Schölmerich J, Herfarth H, Rogler G.

Physiological Role of Macrophage Inflammatory Protein 3-α Induction during Maturation of intestinal Makrophages.

J Immunol, 175:1389-1398, 2005

[19] Andus T, Rogler G, Daig R, et al.

The role of macrophages, Inflammatory Bowel Disease.

Klouwer Academic Publisher, Boston, London, 281-297, 1995

[20] Yao K, Yao T, Iwashita A, Matsui T, Kamachi S.

Microaggragate of Immunostained Macrophages in Noninflamed Gastroduodenal Mucosa: A new Useful Histological Marker for Differentiating Crohn`s Colitis from Ulcerative Colitis.

Am J Gastroenterol, 59:1967-1973, 2000

[21] Grimm MC, Pavli P, Van de Pol E, Doe WF.

Aliterations of the phenotype of colonic macrophages in inflammatory bowel disease.

Eur J Gastroeterol Hepatol, 9(9):893-899, 1997

[23] Rogler G, Hausmann M,Spöttl T, Vogl D, Aschenbrenner E, Andus T, Falk W, Schölmerich J, Goss V.

T-cell co-stimultory molecules are upregulated on intestinal macrophages from inflammatory bowel disease mucosa.

Eur J Gastroenterol Hepatol, 11(10):110-1111, 1999

[24] Hausmann M, Obermeier F, Schreiter K, Spöttl T, Falk W, Schölmerich J, Herfarth H, Saftig P, Rogler G.

Cathepsin D is up-regulated in inflammatory bowel disease macrophages.

Clin Exp Immunol. 136(1): 157-167

[25] Hausmann M, Spöttl T, Andus T, Rothe G, Falk W, Schölmerich J, Herfarth H, Rogler G.

Substractive screening reveals up-regulation of NADPH-oxidase expression in Crohn´s disease intestinal macrophages.

[27] Grimm MC, Elsbury SK, Pavli P, Doe WF.

Enhanced expression and production of monocyte chemoattractant protein-1 in inflammatory bowel disease mucosa.

J Leucoc Biol, 59(6): 804-812, 1996

[28] Spöttl T, Hausmann M, Herlyn M, Gunckl M, Dirmeier A, Falk W, Herfarth H, Schölmerich J, Rogler G.

Monocyte chemoattractant protein-1 (MCP-1) inhibits the intestinal-like differentiation of monocytes.

Clin Exp Immunol, 145(1):190-199, 2006

[29] Meylan E, Tschopp J, Karin M.

Intracellular pattern recognition receptors in the host response.

Nature, 446: 39-44, 2006

[30] Tschopp J, Martinon F, Burns K.

NALPs: A novel protein family involved in inflammation.

Nature Rev Molec Cell Biol. 4:95-104, 2003

[31] Ferrero-Milani L, Nielsen OH, Andersen PS, Girardin SE.

Chronic inflammation: importance of NOD2 and NALP3 in in interleukin-1β generation.

Clin and Exp Immunol. 147: 227-235, 2006

[32] Chamaillard M, Girardin SE, Viala J, Philpott DJ.

Nods, Nalps and Naip: intracellular regulators of bacterial-induced inflammation.

Cell. Microbiol. 5(9):581-592, 2003

[33] Martinon F, Tschopp J.

Inflammatory Caspases, Linking an Intracelllar Innate Immune System to Autoinflammatory Disease.

Cell. 117(5): 561-574, 2004

[34] Petrelli V, Papin S, Tschopp J.

The inflammasome.

Curr. Biol. 15(15): 581-582, 2005

[35] Tschopp J.

Pathogen and danger-sensing platforms that trigger apoptosis and inflammation.

www.unil.ch/ib/page9499.html gesehen am 3.12.2007

[36] Andus T, Daig R, Vogl D, Aschenbrenner E, Lock G, Hollerbach S, Köllinger M, Schölmerich J, Gross V.

Imbalance of the interleukin 1 system in colonic mucosa-association with intestinal inflammation and interleukin 1 receptor agonist genotyp 2.

Gut.41:651-657, 1997

[37] Bode H, Schmitz H, Fromm M, Scholz P, Riecken EO, Schulke JD.

IL-1β and TNF-α, but not IFN-γ, IL-6oder IL-8, are secretory mediators in human distal colon.

CYTOKINE.10(6): 457-465, 1998

[38] Dinarello CA.

Unraveling the NALP-3/IL-1β Inflammasome: A Big Lesson from a Small Mutation.

Immunity, 20:243-246, 2004

[39] Pizarro TT, Michie MH, Bentz M, Woraratanadharm J, Smith MF, Foley E, Moskaluk CA,Bickston JS, Cominelli F.

IL-18, a Novel Immunoregulatory Cytokine, is up-Regulated in Crohn´s Disease: Expression and Localization in Intestinal Mucosal Cells.

The J. of Immun. 162:6829-6835, 1999

[40] Sutterwala FS, Ogura Y, Szczepanik M, Tejero ML, Lichtenberger GS, Grant EP, Bertin J, Coyle A, Galan JE, Askenase PW, Flavell R.

Critical Role for NALP3/CIAS1/ Cryopyrin in Innate and Adaptive Immunity through Its Regulation of Caspase-1.

Immunity,24(3):317-327, 2006

[41] Kanneganti TD, Özören N, Body-Malapel M, Amer A, Park JH, Franchi L, Whitfield J, Barchet W, Colonna M, Vandenabeele P, Bertin J, Coyle A, Grant EP, Akira S, Nunez G.

Bacterial RNA and small antiviral compounds activate caspase-1 through cryopyrin/Nalp3.

Nature, 440:233-236, 2006

[42] Martinon F, Agostini L, Meylan E, Tschopp J.

Identification of Bacterial Muramyl Dipeptide as Activator of the NALP3 Cryopyrin Inflammasome.

Curr Biol. 14:1929-1934, 2004

[43] Inohara N, Ogura Y, Fontalba A, Gutierrez O, Pons F, Crespo J, Fukase K, Inamura S, Kusumoto S, Hashimoto M, Forster S, Moran AP,

Fernandez-Luna JL, Nunez G.

Host Recognition of Bacterial Muramyl Dipeptide Mediated through NOD2.

J Biol Chem, 278(8):5509-5512, 2003

[44] Martinon F.

Detection of humane danger signals by NALP3.

J Leucocyte Biol, 83, 2008

[45] Ogura Y, Sutterwala FS, Flavell R.

The Inflammasome: First Line of the Immune Response to Cell Stress.

Cell, 126, 2006

[46] Bull DM, Bookman MA.

Isolation and Functional Characterization of Human Intestinal Mucosal Lymphoid Cells.

J Clinical Investigation, 59: 966-974, 1977

[47] Hausmann M, Kiessling S, Meistermann S, Webb G, Spöttl T, Andus T, Schölmerich J, Herfarth H, Ray K, Falk W, Rogler G.

Toll-like Receptors 2 and 4 Are Up-regulated During Intestinal Inflammation.

Gastroenterology, 122: 1987-2000, 2002

[48] Mahida YR, Wu K, Jewell DP.

Enhanced production of interleukin 1-beta by mononuclear cells isolated from mucosa with active ulcerative colitis of Crohn`s disease.

[52] Kummer JA, Broekhuizen R, Everett H, Agostini L, Kuijk L, Martinon F, van Bruggen R, Tschopp J.

Inflammasome Components NALP 1 and show Distinct but Separate Expression Profile in Human Tissues Suggesting a Site-specific Role in the Inflammatory Response.

J Histochemistry and Cytochemistry.55 (5): 443-452, 2007

Im Dokument 1.2.1 Morbus Crohn (Seite 66-74)